Summary
One in ten severe acute respiratory syndrome coronavirus 2 infections result in prolonged symptoms termed long coronavirus disease (Covid), yet disease phenotypes and mechanisms are poorly understood. This study profiled 368 plasma proteins in 657 participants ≥3 months following hospitalisation. Of these, 426 had at least one long Covid symptom and 233 had fully recovered.
The study aimed to understand inflammatory processes that underlie Long Covid. The findings suggest that specific inflammatory pathways related to tissue damage are implicated in subtypes of Long Covid, which might be targeted in future therapeutic trials.
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